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Attentrol (Atomoxetine)
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Atomoxetine

Attentrol is used as part of a total treatment program to increase the ability to pay attention and decrease impulsiveness and hyperactivity in children and adults with ADHD. Attentrol is in a class of medications called selective norepinephrine reuptake inhibitors. It works by increasing the levels of norepinephrine, a natural substance in the brain that is needed to control behavior.

Other names for this medication:
Atomoxetine, Strattera, Axepta, Attentrol, Attera,

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Attentrol comes as a capsule to take by mouth. It is usually taken either once a day in the morning, or twice a day in the morning and late afternoon or early evening. Attentrol may be taken with or without food. Take Attentrol at around the same time(s) every day. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Attentrol exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

Swallow Attentrol capsules whole; do not open, chew, or crush them. If a capsule is accidentally broken or opened, wash away the loose powder with water right away. Try not to touch the powder and be especially careful not to get the powder in your eyes. If you do get powder in your eyes, rinse them with water right away and call your doctor.

Your doctor will probably start you on a low dose of Attentrol and increase your dose after at least 3 days. Your doctor may increase your dose again after 2–4 weeks. You may notice improvement in your symptoms during the first week of your treatment, but it may take up to one month for you to feel the full benefit of Attentrol.

Attentrol may help to control the symptoms of ADHD but will not cure the condition. Continue to take Attentrol even if you feel well. Do not stop taking Attentrol without talking to your doctor.

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Photosynthetic Light Utilization Efficiency, Photosystem II Heterogeneity, and Fluorescence Quenching in Chlamydomonas reinhardtii during the Induction of the CO(2)-Concentrating Mechanism. His behaviour progressively improved in parallel with very good recovery of verbal skills. This suggests that oxidized LDL, a newly reported weak agonist, contributes to atherogenesis and thrombogenesis by stimulating platelets. These genes encode a global transcriptional regulator and a basic helix-loop-helix transcription factor, respectively. In this dose escalation study, we evaluated the combination of Genasense and mitoxantrone, a standard chemotherapy for patients with HRPC.

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